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| OPHTHALMIC IMAGE |
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| Year : 2022 | Volume
: 2
| Issue : 2 | Page : 632 |
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Idiopathic intracranial hypertension with peripapillary choroidal neovascular membrane
Devashish Dubey1, Devika Singh2
1 Department of Vitreoretina, Sankara Eye Hospital, Jaipur, Rajasthan, India
2 Department of Cornea, Sankara Eye Hospital, Jaipur, Rajasthan, India
| Date of Web Publication | 13-Apr-2022 |
Correspondence Address:
Devashish Dubey
Sankara Eye Hospital, 6, Central Spine Rd, Sector 2, Sector 6, Vidyadhar Nagar, Jaipur - 302 039, Rajasthan
India
 Source of Support: None, Conflict of Interest: None
DOI: 10.4103/ijo.IJO_2053_21
How to cite this article:
Dubey D, Singh D. Idiopathic intracranial hypertension with peripapillary choroidal neovascular membrane. Indian J Ophthalmol Case Rep 2022;2:632 |
How to cite this URL:
Dubey D, Singh D. Idiopathic intracranial hypertension with peripapillary choroidal neovascular membrane. Indian J Ophthalmol Case Rep [serial online] 2022 [cited 2022 May 18];2:632. Available from: https://www.ijoreports.in/text.asp?2022/2/2/632/342920 |
A 34-year-old lady on treatment for idiopathic intracranial hypertension (IIH) presented with blurring of vision in the left eye for 3 months. Her best-corrected visual acuity was 20/20 and 20/200 in the right and left eye, respectively. Examination revealed papilledema with an orange-red lesion, temporal to the optic disc with subretinal fluid (SRF) in the left eye [Figure 1]a and [Figure 1]b. Fundus fluorescein angiography was suggestive of bilateral disc edema [Figure 1]c and [Figure 1]d along with a peripapillary choroidal neovascular membrane in the left eye (PCNVM) [Figure 1]d and [Figure 1]e. | Figure 1: Color fundus photograph showing bilateral disc edema (a and b). The left eye shows an orange-red lesion of approximately half-disc diameter temporal to the optic disc along with macular hard exudates and subretinal fluid (b). Fundus fluorescein angiography image showing bilateral early disc hyperfluorescence (c and d). The lesion in the left eye showed early hyper-fluorescence in a lacy pattern increasing in size and intensity through the late phase suggestive of a classic CNVM (d and e)
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The patient was treated with an anti-VEGF injection, resulting in resolution of SRF and improvement in vision [Figure 2]a and [Figure 2]b. | Figure 2: Optical coherence tomography (SD-OCT) image showing a subretinal peripapillary hyper-reflective lesion with subretinal fluid (a). Regression of the lesion with complete resolution of subretinal fluid noted after intravitreal bevacizumab injection (b)
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PCNVM is extremely rare in IIH. The prevalence of PCNVM in IIH has previously been reported as 0.53%.[1],[2] Self-involution with treatment for IIH is possible, but anti-VEGF injection is required in vision-threatening diseases.[1],[3]
Financial support and sponsorship
Nil.
Conflicts of interest
There are no conflicts of interest.
| References | |  |
| 1. | Wendel L, Lee AG, Boldt HC, Kardon RH, Wall M. Subretinal neovascular membrane in idiopathic intracranial hypertension. Am J Ophthalmol 2006;141:573-4.  |
| 2. | Lee J, Ferrucci S. Peripapillary subretinal neovascular membranes: A review. Optometry 2011;82:681-8. |
| 3. | Jamerson SC, Arunagiri G, Ellis BD, Leys MJ. Intravitreal bevacizumab for the treatment of choroidal neovascularization secondary to pseudutomor cerebri. Int Ophthalmol 2009;29:183-5. |
[Figure 1], [Figure 2]
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